Search results for " Cellular and Molecular Neuroscience"

showing 10 items of 33 documents

Structural, ultrastructural, and morphometric study of the zebrafish ocular surface: a model for human corneal diseases?

2018

Purpose: A morphological and morphometric study of the adult zebrafish ocular surface was performed to provide a comprehensive description of its parts and to evaluate its similarity to the human. Materials and Methods: The eyes of adult zebrafish were processed for light, transmission and scanning electron microscopy, and for immunohistochemical stain of corneal nerves; a morphometric analysis was also performed on several morphological parameters. Results: The corneal epithelium was formed by five layers of cells. No Bowman’s layer could be demonstrated. The stroma consisted of lamellae of different thickness with few keratocytes. The Descemet’s membrane was absent as the flat and polygon…

0301 basic medicineBowman's layer; corneal nerves; Descemet's membrane; ocular surface; Zebrafish; Ophthalmology; Sensory Systems; Cellular and Molecular NeuroscienceConjunctivaCorneal StromaBiologycorneal nerveCorneal DiseasesCornea03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineMicroscopy Electron TransmissionCorneamedicineAnimalsHumansTrigeminal NerveBowman MembraneZebrafishDescemet MembraneZebrafishTrigeminal nerveocular surfaceBowman’s layerCorneal DiseasesEndothelium CornealEpithelium CornealDescemet’s membraneEpithelial CellsAnatomybiology.organism_classificationSensory SystemsDescemet's membraneOphthalmology030104 developmental biologymedicine.anatomical_structureModels Animal030221 ophthalmology & optometryUltrastructureMicroscopy Electron ScanningGoblet CellsBowman MembraneConjunctivaCurrent eye research
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Gene therapy for chondral and osteochondral regeneration: is the future now?

2017

Gene therapy might represent a promising strategy for chondral and osteochondral defects repair by balancing the management of temporary joint mechanical incompetence with altered metabolic and inflammatory homeostasis. This review analysed preclinical and clinical studies on gene therapy for the repair of articular cartilage defects performed over the last 10 years, focussing on expression vectors (non-viral and viral), type of genes delivered and gene therapy procedures (direct or indirect). Plasmids (non-viral expression vectors) and adenovirus (viral vectors) were the most employed vectors in preclinical studies. Genes delivered encoded mainly for growth factors, followed by transcripti…

0301 basic medicineCartilage ArticularExpression vectorPathologymedicine.medical_specialtyCell signalingCartilage repair; Expression vectors; Gene therapy procedures; Osteoarthritis; Regenerative medicine; Molecular Medicine; Molecular Biology; Pharmacology; Cellular and Molecular Neuroscience; Cell BiologyBone RegenerationInflammatory arthritisGenetic enhancementGene therapy procedureOsteoarthritisViral vector03 medical and health sciencesCellular and Molecular NeuroscienceCartilage repairChondrocytesInterferonSettore BIO/13 - Biologia ApplicataOsteoarthritismedicineAnimalsHumansRegenerationMolecular BiologyPharmacologyExpression vectorbusiness.industryRegeneration (biology)Cell BiologyGenetic Therapymedicine.disease030104 developmental biologyRegenerative medicineCancer researchMolecular MedicineOsteoarthritibusinessmedicine.drugCellular and molecular life sciences : CMLS
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Comparative Performance of Linear Multielectrode Probes and Single-Tip Electrodes for Intracortical Microstimulation and Single-Neuron Recording in M…

2017

Intracortical microstimulation (ICMS) is one of the most widely employed techniques for providing causal evidence of the relationship between neuronal activity and specific motor, perceptual, or even cognitive functions. In recent years, several new types of linear multielectrode silicon probes have been developed, allowing researchers to sample neuronal activity at different depths along the same cortical site simultaneously and with high spatial precision. Nevertheless, silicon multielectrode probes have been rarely employed for ICMS studies and, more importantly, it is unknown whether and to what extent they can be used for combined recording and stimulation experiments. Here, we address…

0301 basic medicineHand regionMaterials scienceCognitive NeuroscienceNeuroscience (miscellaneous)Acute recording; Chronic recording; Electrical stimulation; Macaque monkey; Silicon probes; Neuroscience (miscellaneous); Developmental Neuroscience; Cognitive Neuroscience; Cellular and Molecular NeuroscienceMacaquechronic recording03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineDevelopmental Neurosciencebiology.animalmedicinePremovement neuronal activitysilicon probeselectrical stimulationOriginal Researchbiologytechnology industry and agricultureequipment and suppliesmacaque monkeyMicroelectrode030104 developmental biologyIntracortical microstimulationmedicine.anatomical_structureElectrodeacute recordingNeuron030217 neurology & neurosurgeryBiomedical engineeringMotor cortexNeuroscienceFrontiers in systems neuroscience
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Modulation of the TGF-β1-induced epithelial to mesenchymal transition (EMT) mediated by P1 and P2 purine receptors in MDCK cells

2017

Epithelial to mesenchymal transition (EMT) occurs during embryogenesis or under pathological conditions such as hypoxia, injury, chronic inflammation, or tissue fibrosis. In renal tubular epithelial cells (MDCK), TGF-β1 induces EMT by reducing or increasing epithelial or mesenchymal marker expression, respectively. In this study, we confirmed that the cAMP analogues, 8-CPT-cAMP or N6-Ph-cAMP, inhibited the TGF-β1-driven overexpression of the mesenchymal markers ZEB-1, Slug, Fibronectin, and α-SMA. Furthermore, we showed that A1, A2A, P2Y1, P2Y11, and P2X7 purine receptor agonists modulated the TGF-β1-induced EMT through the involvement of PKA and/or MAPK/ERK signaling. The stimulation o…

0301 basic medicineMAPK/ERK pathwayMadin Darby canine kidney cellEpithelial-Mesenchymal TransitionFibrosiCellTransforming growth factor β1InflammationStimulationBiologyEpithelial to mesenchymal transition; Fibrosis; Madin Darby canine kidney cells; P1/P2 purinergic receptors; Transforming growth factor β1; Molecular Biology; Cellular and Molecular Neuroscience; Cell BiologyTransforming Growth Factor beta103 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineDogsmedicineAnimalsEpithelial–mesenchymal transitionReceptorMolecular BiologyEpithelial to mesenchymal transitionP1/P2 purinergic receptorReceptors Purinergic P2Mesenchymal stem cellReceptors Purinergic P1Cell BiologyMadin Darby canine kidney cellsFibrosisCell biologyFibronectin030104 developmental biologymedicine.anatomical_structure030220 oncology & carcinogenesisbiology.proteinP1/P2 purinergic receptorsOriginal ArticleTransforming growth factor β1medicine.symptomTransforming growth factor
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Flexible multi-beam light-sheet fluorescence microscope for live imaging without striping artifacts

2018

The development of light-sheet fluorescence microscopy (LSFM) has greatly expanded the experimental capabilities in many biological and biomedical research fields, enabling for example live studies of murine and zebrafish neural activity or of cell growth and division. The key feature of the method is the selective illumination of a sample single plane, providing an intrinsic optical sectioning and allowing direct 2D image recording. On the other hand, this excitation scheme is more affected by absorption or scattering artifacts in comparison to point scanning methods, leading to un-even illumination. We present here an easily implementable method, based on acousto-optical deflectors (AOD),…

0301 basic medicineMaterials scienceOptical sectioningNeuroscience (miscellaneous)acousto optic deflectorbrain imagingAcousto optic deflector; Brain imaging; Fast volumetric imaging; Light-sheet fluorescence microscopy; Striping artifacts; Zebrafish; Anatomy; Neuroscience (miscellaneous); Cellular and Molecular Neurosciencelight-sheet fluorescence microscopy striping artifacts fast volumetric imaging acousto optic deflector brain imaging zebrafishfast volumetric imaginglcsh:RC321-571lcsh:QM1-69503 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineOpticsLive cell imagingFluorescence microscopeTechnology ReportAbsorption (electromagnetic radiation)lcsh:Neurosciences. Biological psychiatry. Neuropsychiatrybusiness.industryScatteringlcsh:Human anatomyzebrafishSample (graphics)striping artifactsAcousto optic deflector Brain imaging Fast volumetric imaging Light-sheet fluorescence microscopy Striping artifacts Zebrafish Anatomy Neuroscience (miscellaneous) Cellular and Molecular Neurosciencelight-sheet fluorescence microscopy030104 developmental biologyFeature (computer vision)Light sheet fluorescence microscopyAnatomybusiness030217 neurology & neurosurgeryNeuroscience
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The Role of Anabolic Androgenic Steroids in Disruption of the Physiological Function in Discrete Areas of the Central Nervous System

2017

: Anabolic-androgenic steroids (AAS) abuse is often associated with a wide spectrum of adverse effects. These drugs are frequently abused by adolescents and athletes for esthetic purposes, as well as for improvement of their endurance and performances. In this literature review, we evaluated the correlation between AAS and anxiety or aggression. Two pathways are thought to be involved in AAS-induced behavioral disorders. Direct pathway via the amygdalo-fugal pathway, which connects the central nucleus of the amygdala to the brainstem, is involved in cognitive-emotive and homeostatic processes. The latter is modified by chronic AAS use, which subsequently leads to increased anxiety. Indirect…

0301 basic medicineNeuroscience (miscellaneous)SerotonergicAbuse; Amygdala; Anabolic-androgenic steroids (AAS); Behavioral disorders; Central nervous system; Molecular mechanisms; Cellular and Molecular NeuroscienceAmygdalaAbuseMolecular mechanism03 medical and health sciencesCellular and Molecular NeuroscienceAnabolic Agents0302 clinical medicinemedicineAnimalsHumansDirect pathway of movementAggressionCentral nucleus of the amygdalaDopaminergicMolecular mechanismsAmygdalaAggressionBehavioral disorders030104 developmental biologymedicine.anatomical_structureNeurologyHypothalamusCentral nervous systemBehavioral disorderAndrogensAnxietySteroidsmedicine.symptomPsychologyNeuroscience030217 neurology & neurosurgeryAnabolic-androgenic steroids (AAS)
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Parkinson's disease: towards better preclinical models and personalized treatments.

2016

Non peer reviewed

0301 basic medicineParkinson's diseaseeducationMEDLINEBioinformatics03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineDEFICITSMedicineAnimalsHumansMolecular BiologyPharmacologybusiness.industryParkinson DiseaseCell Biologymedicine.diseaseMolecular medicine3. Good healthMICE030104 developmental biologyNeuroprotective AgentsCell Biology; Molecular Biology; Molecular Medicine; Pharmacology; Cellular and Molecular NeuroscienceMolecular Medicine3111 Biomedicinebusiness030217 neurology & neurosurgeryCellular and molecular life sciences : CMLS
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Introducing the concept of “CSF-shift edema” in traumatic brain injury

2018

Brain edema after severe traumatic brain injury (TBI) plays an important role in the outcome and survival of injured patients. It is also one of the main targets in the therapeutic approach in the current clinical practice. To date, the pathophysiology of traumatic brain swelling is complex and, being that it is thought to be mainly cytotoxic and vasogenic in origin, not yet entirely understood. However, based on new understandings of the hydrodynamic aspects of cerebrospinal fluid (CSF), an additional mechanism of brain swelling can be considered. An increase in pressure into the subarachnoid space, secondary to traumatic subarachnoid hemorrhage, would result in a rapid shift of CSF from t…

0301 basic medicinePathologymedicine.medical_specialtySubarachnoid hemorrhageTraumatic brain injurybrain edema; cisternostomy; decompressive hemicraniectomy; paravascular pathway; traumatic brain injury; Cellular and Molecular NeuroscienceBrain water03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineCerebrospinal fluidEdemaBrain Injuries TraumaticmedicineHumansparavascular pathwaybrain edemaBrain edemabusiness.industrytraumatic brain injurymedicine.diseasecisternostomyPathophysiology030104 developmental biologymedicine.anatomical_structureSubarachnoid spacemedicine.symptomExtracellular Spacebusinessdecompressive hemicraniectomybrain edema; cisternostomy; decompressive hemicraniectomy; paravascular pathway; traumatic brain injury030217 neurology & neurosurgeryJournal of Neuroscience Research
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Linoleic acid: Is this the key that unlocks the quantum brain? Insights linking broken symmetries in molecular biology, mood disorders and personalis…

2017

Abstract In this paper we present a mechanistic model that integrates subneuronal structures, namely ion channels, membrane fatty acids, lipid rafts, G proteins and the cytoskeleton in a dynamic system that is finely tuned in a healthy brain. We also argue that subtle changes in the composition of the membrane’s fatty acids may lead to down-stream effects causing dysregulation of the membrane, cytoskeleton and their interface. Such exquisite sensitivity to minor changes is known to occur in physical systems undergoing phase transitions, the simplest and most studied of them is the so-called Ising model, which exhibits a phase transition at a finite temperature between an ordered and disorde…

0301 basic medicinePhase transitionLinoleic acidMood DisorderModels NeurologicalPhysical systemAntidepressantContext (language use)MicrotubuleReviewlcsh:RC321-57103 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineAntidepressants; Cytoskeleton; Depression; Ion channels; Ising model; Linoleic acid; Lipid raft; Microtubule; Mood disorders; Quantum states; Linoleic Acid; Mood Disorders; Brain; Models Neurological; Neuroscience (all); Cellular and Molecular NeuroscienceIsing modelCytoskeletonlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryLipid raftQuantumIon channelCytoskeletonNeuroscience (all)ChemistryDepressionGeneral Neurosciencelcsh:QP351-495BrainQuantum statesMood disorders Linoleic acid Ion channels Cytoskeleton Microtubule Lipid raft Depression Antidepressants Ising model Quantum statesAntidepressantsQuantum stateLipid raftlcsh:Neurophysiology and neuropsychology030104 developmental biologyIon channelsMood disordersIsing modelIon channelNeuroscience030217 neurology & neurosurgery
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Usherin defects lead to early-onset retinal dysfunction in zebrafish

2018

Mutations in USH2A are the most frequent cause of Usher syndrome and autosomal recessive nonsyndromic retinitis pigmentosa. To unravel the pathogenic mechanisms underlying USH2A-associated retinal degeneration and to evaluate future therapeutic strategies that could potentially halt the progression of this devastating disorder, an animal model is needed. The available Ush2a knock-out mouse model does not mimic the human phenotype, because it presents with only a mild and late-onset retinal degeneration. Using CRISPR/Cas9-technology, we introduced protein-truncating germline lesions into the zebrafish ush2a gene (ush2a(rmc1): c.2337_2342delinsAC; p.Cys780GlnfsTer32 and ush2a(b1245): c.15520_…

0301 basic medicineRetinal degenerationGenotyping TechniquesUsher syndrome2804 Cellular and Molecular NeuroscienceApoptosis030105 genetics & heredityBiologyArticleRetinaGermlineSensory disorders Donders Center for Medical Neuroscience [Radboudumc 12]Gene Knockout Techniques03 medical and health sciencesCellular and Molecular NeuroscienceUSH2 complex2809 Sensory SystemsAll institutes and research themes of the Radboud University Medical CenterRetinitis pigmentosaElectroretinographymedicineotorhinolaryngologic diseasesJournal ArticleAnimalsMicroscopy ImmunoelectronZebrafishZebrafishExtracellular Matrix ProteinsRetinal DegenerationMembrane ProteinsZebrafish ProteinsRetinal Photoreceptor Cell Outer Segmentmedicine.diseasebiology.organism_classification2731 OphthalmologySensory Systems10124 Institute of Molecular Life SciencesCell biologyDisease Models AnimalOphthalmology030104 developmental biologyGene Expression RegulationEctodomainMutation570 Life sciences; biologyXenotropic and Polytropic Retrovirus ReceptorUsher SyndromesErg
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